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GHK-Cu 50mg
Anti-Aging & Longevity
ANTI-AGING

Buy GHK-Cu 50mg

Copper tripeptide for skin regeneration, wound healing, and anti-aging gene activation

Buy GHK-Cu 50mg — GHK-Cu 50mg — the accessible entry point for the copper tripeptide that activates thousands of anti-aging genes and restores skin collagen, elastin, and barrier function.

  • Stimulates collagen I, III, and V synthesis in dermal fibroblasts
  • Activates thousands of anti-aging genes in skin and systemic tissue
  • Reduces fine lines and wrinkles through collagen restoration
  • Accelerates wound healing and skin barrier repair
  • Improves skin tone, thickness, and hydration
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Buy GHK-Cu 50mg on Phiogen

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GHK-Cu 50mg: The Copper Tripeptide Entry Point for Skin Regeneration and Anti-Aging Gene Activation

50mg Starting Supply

The ideal introductory quantity for users new to GHK-Cu — sufficient for a full 8-week injectable protocol or months of topical serum application.

Collagen Restoration

Stimulates dermal fibroblasts to produce all major collagen types — rebuilding the structural protein network that gives skin its thickness, strength, and wrinkle resistance.

Dual-Route Efficacy

Works topically on skin structure and injected for systemic anti-aging gene activation — a rare peptide that delivers genuine value through both administration routes.

Fifty Years of Research

Discovered in 1973, GHK-Cu has one of the longest research histories of any cosmeceutical peptide — providing decades of safety and efficacy data.

The Science Behind GHK-Cu 50mg

The Original Copper Peptide

GHK-Cu (glycyl-L-histidyl-L-lysine copper) was first isolated by Loren Pickart in 1973 from human plasma albumin. Decades of subsequent research revealed that this simple tripeptide coordinates copper ions and serves as a master regulator of tissue remodeling, wound healing, and anti-aging gene expression. The 50mg vial is the ideal starting point for users beginning topical or injectable GHK-Cu protocols.

Gene Expression and Skin Rejuvenation

At the molecular level, GHK-Cu acts as a transcription factor activator, modulating the expression of genes controlling collagen synthesis, matrix metalloproteinase regulation, antioxidant defense, and stem cell activation. Applied to skin, it stimulates dermal fibroblasts to produce collagen I, III, and V, restores elastin and proteoglycan content, and activates the glycosaminoglycan synthesis that maintains skin hydration and structure.

Multiple Administration Routes

GHK-Cu is effective both topically and via injection. Topically at 0.1–2%, it reduces fine lines, improves skin tone, and accelerates wound healing. Injectable GHK-Cu at lower concentrations (0.1–0.5mg subcutaneous) produces systemic gene activation effects — anti-inflammatory, neuroprotective, and antioxidant — that topical application cannot match in scope.

Complete GHK-Cu 50mg Benefits

  • Stimulates collagen I, III, and V synthesis in dermal fibroblasts
  • Activates thousands of anti-aging genes in skin and systemic tissue
  • Reduces fine lines and wrinkles through collagen restoration
  • Accelerates wound healing and skin barrier repair
  • Improves skin tone, thickness, and hydration
  • Anti-inflammatory via NF-κB modulation
  • Compatible with both topical and injectable administration

GHK-Cu 50mg Dosing Protocol

Topical: Dissolve 50mg in 50mL carrier (peptide serum base). Apply 2x daily.

Injectable: Reconstitute in bacteriostatic water at 1–5mg/mL. Dose 0.1–0.5mg subcutaneous daily.

Cycle: Topical — continuous; Injectable — 4–8 week cycles

All information on this site is for educational purposes only. Always consult with a qualified healthcare provider before use. COA documentation is available for all products.

GHK-Cu 50mg

COA verified · Third-party tested · US domestic shipping

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$53.99
$59.99
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High-volume copper tripeptide for skin regeneration, anti-aging, and wound repair

Verified GHK-Cu 100mg — GHK-Cu 100mg — maximum supply of the copper-binding tripeptide that activates over 4,000 human genes, restores skin structure, and reverses markers of cellular aging.

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